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01-31-2002, 05:55 PM | #11 |
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No, you silly evilutionists...
Clearly, these genes are the remnants of DEGRADED genomes, since information can only be LOST by mutations. Obviously, insects HAD circulatory systems and clotting blood BEFORE THE FALL!!! And the probably lived far longer lives as a result... and they were much harder to squash, but they didn't bother anyone because they used their stings and bites only on PLANTS... they were all vegetarian bugs back then, and yeast needed insulin to regulate their blood sugar because they couldn't decompose dead things... because NOTHING DIED - the Creation was PERFECT!!! So the yeast politely sipped sugary fruit juice like all the other perfect organisms in the Garden of Eden, so they needed insulin to regulate all that sugar. Got it? [ January 31, 2002: Message edited by: wehappyfew ]</p> |
01-31-2002, 07:09 PM | #12 | |
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02-01-2002, 06:47 AM | #13 | |
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02-01-2002, 07:02 AM | #14 | ||
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02-01-2002, 08:46 AM | #15 | |
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See: Cell 1992 Mar 20;68(6):1077-90 Unipolar cell divisions in the yeast S. cerevisiae lead to filamentous growth: regulation by starvation and RAS. Gimeno CJ, Ljungdahl PO, Styles CA, Fink GR. Diploid S. cerevisiae strains undergo a dimorphic transition that involves changes in cell shape and the pattern of cell division and results in invasive filamentous growth in response to starvation for nitrogen. Cells become long and thin and form pseudohyphae that grow away from the colony and invade the agar medium. Pseudohyphal growth allows yeast cells to forage for nutrients. Pseudohyphal growth requires the polar budding pattern of a/alpha diploid cells; haploid axially budding cells of identical genotype cannot undergo this dimorphic transition. Constitutive activation of RAS2 or mutation of SHR3, a gene required for amino acid uptake, enhance the pseudohyphal phenotype; a dominant mutation in RSR1/BUD1 that causes random budding suppresses pseudohyphal growth. |
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02-01-2002, 08:53 AM | #16 | |
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