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#141 | |
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I think it was probably a combination of things but I think that the most unique factor is that we were the first complex animal to feed on sunlight. I believe in the aquatic ape theory and that we are beach monkeys who got a lot of sun and when push came to shove we returned back to life's original food source. I think our skin can/could produce more then just vitamin d out of sunlight if it had to. Those days may be gone but I think that we really were the perfect animal for a little while. I think it was just a matter of time before nature came up with an animal that could feed on the sun to. Plant's don't have the aging thing that animals do and I think for some reason that eating what they ate stopped our aging to. |
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#142 | |
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I thought you were having a problem with cellular communication of genetic material. |
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#143 | ||
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:wave: Bye bye now, crazy person. ::walk away slowly. don't turn your back. OK, now RUN! :: Folks, I think it is now clear that there is no point in discussing science with Elijah. His understanding is at the same level as his primary reading material, and he does not make a disctinction between insanity and sense. |
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#144 | ||
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Fortunately, he's well matched with his "girl" Nimi, who says aging doesn't cause wrinkles..."chronological aging " does. ![]() I can only hope they don't breed more of these. |
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#145 | ||
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mung bean raises an interesting question, though: if the above is true, how do you explain vampires? |
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#146 | ||
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#147 | ||
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... okaaaaay. Well, you're right: It is crazy. It's also not any Lamarkian mechanism for longevity that makes any sense- but, if one believes that humans could once feed on sunlight, also believing that it made them live for millenia is not THAT surprizing. Bye now. (You are a Loki troll, aren't you? Noone can be that crazy.) |
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#148 | |
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#149 |
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This thread has evolved, in a purely Lamarkian fashion, right out of its original forum.
--Coragyps as moderator |
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#150 |
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Interestingly, the alcohol dehydrogenase mutation that is normally associated with the "Asian Flush" involves Glu487 changing to a Lys. IFF the epigenetic modification occurs on the noncoding stand, then epigenetics followed by a normal mutation (methylated Cs become Ts like nobody's business) could be presented as the initial mechanism. As in, those with the silenced C would have a selective advantage if that C mutated to a T.
Now, this is not only anecdotal, but it also has a problem -- it is presently believed (based on other primates) that humans started out *not* being able to metabolize alcohol properly (like the Asians), however, since Europeans lived in routinely filthy conditions, there was a strong selective pressure to be able to metabolize alcohol since it was the most readily available source of potable water (as in, if you couldn't drink alcohol, you would get cholera and die). This wasn't a problem in Asia because Asians drink tea, which is boiled (back in the day, anyway. Bitter tea is better than liquid death!) and has antimicrobial properties. So, it would seem like the actual mutation wouldn't be glu to lys, but rather lys to glu. If the non-coding stand C were methylated and became a T, a silent mutation would result and you'd still have lys. So, I'll go and reject it. |
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