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#801 |
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Hi Principia. Acknowledged.
There is value to me in knowing their arguments, and yes - it isn't a positive presentation of creation. Thus far, the assertion that all radiometric dating has huge flaws is falling on its face. I see CD accepts the age of the earth is > 4 billion years anyway, so it's on to the assertion that there are not gradual evolutionary fossil forms in successive layers of strata. From a layman's view it seems obvious to me the simple organisms are on the bottom, eh? I find the horse literature pretty convincing so far... |
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#802 | |
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#803 | ||
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#804 | ||||||
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What is interesting, as we discussed, is the erroneous grouping of the chimp and gorilla; and especially the fact that it is supported by 100% bootstrap probability. They had no choice but to resort to the just-so polymorphism story with random sorting. What is the probability of that is giving you 100% bootstrap probability? Quote:
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#805 | ||||
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i.e. the amount of mutations in favour of the phylogeny far outweighed those that weren't (the convergent ones) even so much as to have 100 percent bootstrap probability this is how phylogeny works CD - you don't select the positions you want to construct whatever phylogeny you want - you sum over all of them - because the random nature of mutations demands that phylogenies are supported in a statistical rather than absolute sense Quote:
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CD, its not the mutation rate that is different. Its the rate of fixation of mutations Oda et al found exactly what they were expecting the reason that the nonsynonymous mutations have accumulated as rapidly as the synonymous mutations in the hominids is because their sequences are neutral - nonsynonymous mutations make no difference to fitness, and so are fixed at the same rate as synonymous mutations the reason that the same isn't the case in the old world monkeys and the new world monkey is that their urate oxidase still functions -> so the nonsynonymous sites are under selective constraint -> changes in nonsynonymous sites are not fixed at the same rate as synonymous sites, because they have phenotypic consequences so the difference is in the selection on the sequences - not the mutation rates of the sequences - this is confirmation of the fact that the urate oxidase pseudogene in hominids is useless, and its why they looked at nonsynonymous/synonymous rates of substition |
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#806 | |||||||||
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Charles:
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Evolution is a scientific theory which accounts for the fact of common descent. If there was no visible evidence of common descent, there wouldn't be any theory to account for it: just as there wouldn't be a theory of gravity if we were all floating about weightlessly in a hollowed-out asteroid with all memory of our planetary origin erased by mischievous aliens. Why bother to speculate about a parallel Universe in which the evidence for common descent doesn't exist? That isn't the Universe we inhabit. Quote:
And why do you still resort to such obfuscation? Quote:
See my earlier post on this. Quote:
There's also a very curious use of tenses here: "whether evolution can sustain the failure to find...". You should be using the conditional tense when discussing your fantasy Universe: "whether evolution COULD sustain a failure to find...". There is no ACTUAL "failure to find any vestigial structures". You seem to be having difficulty keeping reality and fantasy separate. Quote:
A vestigial organ is one whose ORIGINAL function is obvious from its structure, and from comparisons with otherwise similar organisms. The organ is no longer adequate for its ORIGINAL function, and any additional function is plainly incidental, because (in most cases) the organ is poorly "designed" for its new function also. The human appendix is a classic example of a vestigial organ: there is no reason at all why a dead-end section of intestine should be "designed in" as a site for a concentration of immune-system defenses (but, conversely, a good reason for extra defenses at a site that represents a dangerous avenue of infection). Quote:
That is a rather startling admission of ignorance from one who otherwise claims to know more about evolution than professional biologists! I could explain it for you, but first I want to check that this isn't another bizarre "let's pretend to be stupid" ploy. My apologies in advance if you are genuinely unaware of this. Quote:
...But, if you're using another meaning for "evolution" here, please explain this. Quote:
It is the staggering totality of the fossil record, and the "Tree of Life" of cladistics, that makes the evidence so overwhelming. Thousands upon thousands of species forming a nested hirearchy, supported by millions of fossils confirming the pattern of development of that same hirearchy. And genetics is providing new support of the same hirearchy: again, it is the totality of the genome similarity that matters. Individual genes can be tweaked by mutation in unpredictable ways, but the overall pattern of relationships (based on the percentage difference between the genome of one species and that of another) correlates with the same "Tree of Life" derived from other sources. I am not aware of anything else in science that is better supported (other than obvious observations such as "the Sun emits light"). The odds against this correlation being "accidental" are truly astronomical: the number of possible permutations involved dwarfs such numbers as the number of atoms in the Universe, for instance. Quote:
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#807 |
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This thread is going to be closed later today.
At this point, it is just going around and around in circles, and lacks any kind of focus. The issues being discussed now were also discussed 20 pages ago. I recommend that if anyone still wants to persist in this, that they start a new and much more specifically titled thread. |
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#808 | |
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(Seriously: good move. It has long since been imitating the fabled Oozlum Bird.) Oolon |
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#809 | |||||
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Let me say something else that, from reading your posts, I gather needs saying. You keep on referring to the inactivating mutations common to the gibbons, and to the human, chimp, gorilla, and orangutan; as though they are great coincidences explainable only via common descent. You seem to be missing the fact that they have been carefully culled from a larger set of mutations, which are all over the map. Most of them do not explain the gene inactivation, but if you look hard enough you find one that is common to the human, chimp, gorilla, and orangutan; and you find two that are common to the gibbons. So these are identified by evolutionists as the probable inactivating mutations. OK, fine. But from a non common descent view, if you have a set of mutations and if there are mutational hotspots (an empirical fact), then it is not surprising that (a) they generally will agree with the consensus phylogeny, and (b) amongst all those mutations you will find some that occur in all sequences in a group. The mutational hotspots would be species specific most often; but sometimes specific at higher levels. This is a perfectly reasonable explanation and not, as you have been saying, incredibly unlikely. |
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#810 | |
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Furthermore, your theory of evolution does not require such a hierarchy, so it is hardly so compelling as you suggest. |
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